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A dual role for complement in allergic asthma

Kohl J, Willis-Karp M2007Current Opinion in PharmacologyReview
10.1016/j.coph.2007.01.005PubMed
Immune/InnateNeurologicalRespiratory/Sinus

Abstract

The complement (C) system plays important roles in host defence but activation at inappropriate sites or to an excessive degree can cause host tissue damage. C has been implicated as a factor in the causation or propagation oftissue injury in numer- ous diseases. The brain is an immunologically isolated sitc, sheltered from circulating cells and proteins of the immune sys- tem; nevertheless, there is a growing body of evidence implicat- ing C in numerous brain discascs. In this brief articlc we rcvicw the evidence suggesting a role for C in diseases of the central and peripheral nervous system and discuss the possible sources of C at these sites. Some brain cells synthesise C and also express specific receptors; some are exquisitely sensitive to the lytic effects of C. The evidence suggests that C syr,thesis and activation in the brain are important in immune defence at this site bclt may also play a role in brain disease. .................

Key Biomarkers

C3a anaphylatoxinComplement system activationIgE-mediated response

Symptom Clusters

Allergic asthmaDemyelinationNeurodegenerationNeuroinflammation

References (13)

  • The role of complement in danger sensing and transmissionImmunologic Research · 2006
  • Expression of the Complement Anaphylatoxin C3a and C5a Receptors on Bronchial Epithelial and Smooth Muscle Cells in Models of Sepsis and AsthmaThe Journal of Immunology · 2001
  • Pharmacological Targeting of Anaphylatoxin Receptors during the Effector Phase of Allergic Asthma Suppresses Airway Hyperresponsiveness and Airway InflammationThe Journal of Immunology · 2005
  • Requirements for C5a receptor-mediated IL-4 and IL-13 production and leukotriene C4 generation in human basophilsThe Journal of Immunology · 2000
  • A regulatory role for the C5a anaphylatoxin in type 2 immunity in asthmaJournal of Clinical Investigation · 2006
  • Inhibition of complement activation decreases airway inflammation and hyperresponsivenessAmerican Journal of Respiratory and Critical Care Medicine · 2003
  • Expression of the anaphylatoxin receptors C3aR and C5aR is increased in fatal asthma

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