Correction of central nervous system metabolic abnormalities, deficits in executive cognitive functioning and elevated C4a: a clinical trial using low dose erythropoietin in patients sickened by exposure to water-damaged buildings. IACFS, Fort Lauderdale, Florida
CardiovascularEndocrine (ADH/ACTH/MSH)GastrointestinalHematologicalHypothalamic-PituitaryImmune/InnateNeurologicalRespiratory/Sinus
Summary
No abstract is available for this paper. It was published. CIRS Nexus tags it under Water-Damaged Buildings (WDB), Cardiovascular, Endocrine (ADH/ACTH/MSH), Gastrointestinal, Hematological, Hypothalamic-Pituitary, Immune/Innate, Neurological, Respiratory/Sinus.
Part of Mold & water-damaged buildings research, CIRS biomarkers research, HLA-DR haplotypes research · Curated by Lauren Lowenstein, The Biotoxin Lady
Key Biomarkers
ACTH/cortisolADH/osmolalityAnti-cardiolipin antibodiesAntigliadin antibodiesC4aGlutamate/Glutamine ratioHLA DR haplotypesLactateMMP-9TGF-β1VEGFVIPvon Willebrand factorα-MSH
Symptom Clusters
Capillary hypoperfusionCNS metabolic abnormalitiesCognitive impairmentExecutive dysfunctionMulti-system multi-symptom illnessVisual contrast sensitivity deficits
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