Sequential upregulation of innate immune responses during acute acquisition of illness in patients exposed prospectively to water-damaged buildings. ASTMH, Philadelphia, PA
CardiovascularEndocrine (ADH/ACTH/MSH)GastrointestinalHematologicalHypothalamic-PituitaryImmune/InnateNeurologicalRespiratory/Sinus
Bacterial EndotoxinsIndoor Mold (Stachybotrys, Aspergillus, etc.)MycotoxinsWater-Damaged Buildings (WDB)
Summary
No abstract is available for this paper. It was published. CIRS Nexus tags it under Bacterial Endotoxins, Indoor Mold (Stachybotrys, Aspergillus, etc.), Mycotoxins, Water-Damaged Buildings (WDB), Cardiovascular, Endocrine (ADH/ACTH/MSH), Gastrointestinal, Hematological, Hypothalamic-Pituitary, Immune/Innate, Neurological, Respiratory/Sinus.
Part of Endotoxins research, Mold & water-damaged buildings research, Mycotoxins research, CIRS biomarkers research, HLA-DR haplotypes research, MARCoNS research · Curated by Lauren Lowenstein, The Biotoxin Lady
Key Biomarkers
ACTH/cortisolADH/osmolalityAnticardiolipin antibodies (ACLA)Antigliadin antibodies (AGA)C4aHLA DR haplotypesMARCoNSMMP-9MR spectroscopy (lactate, G/G ratio)TGF-β1VEGFVIPVisual Contrast Sensitivity (VCS)von Willebrand Factor (vWF)α-MSH
Symptom Clusters
Capillary hypoperfusionCognitive dysfunctionEpistaxisHemoptysisMulti-system, multi-symptom illnessNeurologic symptoms
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